Pathology & Parasitology for Veterinary Technicians, 2nd by Leland S. Shapiro

By Leland S. Shapiro

Pathology and Parasitology for Veterinary Technicians, 2E is the booklet pros and destiny execs flip to for distinct details on pathology and parasitology in veterinary medication. It gains uncomplicated descriptions, case reviews, and methodologies for the sensible atmosphere. Veterinary execs and people within the animal, equine, dairy, and chicken fields will make the most of reasons approximately technical techniques, parasite existence cycles, zoonotic power, prognosis, and remedy. A overview of present literature presents invaluable historical past, and the accompanying CD-ROM snapshot Library positive aspects broad colour photographs of parasites and diverse pathologies.

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Certain compounds introduced into the bloodstream can cause this oxidation. The iron in hematin is Feϩ3 instead of Feϩ2 (ferrous iron). The Feϩ3 (ferric iron) does not combine with oxygen, forming methemoglobin instead of hemoglobin. Certain chemicals (nitrates, chlorates) also produce methemoglobinemia. Cattle grazing highly fertilized forage develop nitrate poisoning and cause the formation of methemoglobin. Livestock consuming certain weed killers that contain chlorates do so as well. • Porphyrins are pigments found in both animal and plant life which can make urine, bones, tissues, and teeth red in appearance.

Its purpose in mammals is protection against solar ultraviolet radiation. In lower vertebrates, it is thought to be protective in inflammation. • Lipochromes (lipofuscin and ceroid). These consist of a yellow-brown pigment made from the breakdown of fatty acids and cell membranes. Lipofuscins increase with advancing age and tend to develop in neurons, thyroid epithelium, and muscle cells. One autosomal recessive congenital disorder (neuronal ceroid lipofuscinosis) is seen in cattle, sheep, cats, dogs, and humans.

The black Lab also had pale mucous membranes. The owner had noticed a container of rat poison chewed open in her garage, which the dog had access to from the dog run. The owner believed it must have happened the previous night. Rodenticides of the first generation, such as warfarin (coumarin) or diphacinone, can depress the production of the clotting factors (factors I, II, VII, IX, and X) in the liver for 7 to 10 days. The second generation, such as bromadiolone, can depress the clotting factors for 3 to 4 weeks.

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