Barthelemy-Grino architectes: Frameworks / Trait pour trait by Mohsen Mostafavi (auth.), Brina Goldfarb, Emilie Gourdet

By Mohsen Mostafavi (auth.), Brina Goldfarb, Emilie Gourdet (eds.)

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Inflamm Bowel Dis. 2011;17:1–5. 13. Lesage S, Zouali H, Cezard JP, et al. CARD15/NOD2 mutational analysis and genotype-phenotype correlation in 612 patients with inflammatory bowel disease. Am J Hum Genet. 2002;70:845–57. 14. Pauleau AL, Murray PJ. Role of nod2 in the response of macrophages to toll-like receptor agonists. Mol Cell Biol. 2003;23:7531–9. 15. Kobayashi KS, Chamaillard M, Ogura Y, et al. Nod2-dependent regulation of innate and adaptive immunity in the intestinal tract. Science. 2005;307:731–4.

BMJ. 1996;312:95–6. 11. Tysk C, Lindberg E, Jarnerot G, Floderus-Myrhed B. Ulcerative colitis and Crohn’s disease in an unselected population of monozygotic and dizygotic twins. A study of heritability and the influence of smoking. Gut. 1988;29:990–6. 12. Brant S. Update on the heritability of inflammatory bowel disease: the importance of twin studies. Inflamm Bowel Dis. 2011;17:1–5. 13. Lesage S, Zouali H, Cezard JP, et al. CARD15/NOD2 mutational analysis and genotype-phenotype correlation in 612 patients with inflammatory bowel disease.

Full activation of T cells takes 4–5 days and is accompanied by clonal expansion and remarkable changes in the homing behavior of these cells. Once activated, these effector T cells leave the lymph node returning to the circulation, thus allowing a locally generated immune response to promote defense at distance throughout the gastrointestinal tract [47]. Activated T cells express unique adhesion molecules that direct the cells to sites of inflammation, and indeed T cells primed by mucosal 17 complex class II; Ag antigen; TCR T cell receptor; CTLA-4 cytotoxic T lymphocyte antigen-4 Fig.

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